no place like home…

I am now 6 weeks into my 6 month anaesthetic secondment. There have been some interesting challenges settling into the new job but I am largely enjoying my time perfecting basic airway manoeuvers, laryngoscopy and playing with some brilliant airway toys (McGrath video laryngoscopes, the AirTraq, intubating LMAs etc). I thought I’d share with you a case (from Anaesthetic week 2) that presenting some multifaceted challenges & several points of reflection …

The Case.

A 59 year old male undergoes an elective radical prostatectomy. He is previously well, however takes some ‘herbal Chinese medicines’ that he stopped 2 weeks prior to surgery. His surgery appears to go without a hitch, except for the 1200mL of blood in the surgical suction container at the end of the case. He has received 2 liters of Hartmann’s & 500mL Volvuven during his OT time. He is extubated and taken to recovery at the end of the case where he reports feeling quite comfortable.

I am called back to recovery about 20-25 minutes later to address his hypotension.

On return to recovery, he looks pretty horrible. He is pale & clammy with cool hands. His pulse rate is 95 (sinus rhythm) with a blood pressure of 82/40. I give him two boluses of fluid (500mL each) & his BP promptly improves to 105 systolic….

…..If only it was that simple! I am called 10 minutes later for further hypotension. 80’s on 40’s again…. This time I take a Hemocue which shows a Hb of 68 (was 128 pre-op).

      • 4 units of PRBCs are cross-matched; 2 units given stat
      • repeat Hb 84.
      • Surgeon’s asked to review [blames Chinese medicines, mutters something about post-anaesthetic hypotension, venous oozing & need for further resuscitation, heads back to do the next cystoscopy]…

By this time my boss perches me in recovery to keep a permanent eye on this fella whom I am confident has haemorrhagic shock…

Morning becomes afternoon;

      • Ongoing episodes of hypotension, responding to fluids
      • Declining urine output.
      • Patient remains clammy and at times frankly diaphoretic
      • Bedside USS showed a flat IVC and a hyperdynamic left-ventricle. There was free fluid (~0.6cm) in Morrison’s pouch as well as over the diaphragmatic surface of the liver & spleen…
      • Hb drops back to 60.
      • Massive transfusion formally commenced… (he needs to go back to theatre)
      • Anaesthetic consultant agrees… Urology registrar notified again & again (no action, very reluctant to consider a return to theatre)…

Crisis precipitates action;

      • With ongoing periods of hypotension, our patient eventually reaches a peri-arrest state following an episode of abdominal pain and vomiting, with altered mental state and a systolic BP of 50-something.
      • Anaesthetic consultant and fellow join me at the bedside…
      • MTP continues….
      • Formal repeat bloods show worsening metabolic acidosis with acute kidney injury and hyperkalaemia.
      • Sick of waiting for the registrar to take action, I call the Urologist himself & we finally get a decision to go back to theatre for exploration.

Not over yet;

      • He has one of the more scary inductions I have witnessed.
          • 2mg Midazolam, 100mcg Fentanyl & Cisatracurium (he had received Sugammadex for reversal at the end of his first surgery).
          • This was enough to drop his BP from 130 –> 65 mmHg.
      • He has a large pelvic haematoma evacuated and a further 1500mL of blood in his suction container.
      • Before leaving to theatre (destination: ICU) he has received;
          • 12 units RBC
          • 12 units FFP
          • 2 pools of PLTs
          • 18 units of cryoprecipitate
          • Calcium (20mmol gluconate)
          • Tranexamic acid….

The Reflection

This case was obviously frustrating for a lot of people involved (and incredibly emotional  & exhausting for the recovery staff who provided him with so much time and attention). My subsequent reflection on this scenario has lead to a few realisations….

1) You are a much stronger performer in your own environment….

      • This diagnosis was easy. There was very little else that could have explained this patients’ clinical state, but I appeared to doubt my diagnosis of haemorrhagic shock over & over. Was this because I was in the post-op setting now ?? Bleeding is bleeding….
      • I was obviously uncomfortable in the new surroundings, particularly when it came to escalating my concerns for this patient and advocating for his return to the OT. Perhaps I was afraid of ‘ruffling to many feathers’. I strongly believe that in the face of a registrar’s inaction I would have called a Consultant sooner had I been in my ED.
      • I have no doubt that if this scenario took place  on my home-turf of the ED resus bay, that my assertiveness and push for action would have been with a louder and more confident voice.

2) There is no place like home….

      • When the proverbial hit the fan and this guy decompensated I quickly realised that I had absolutely no idea where the resuscitation equipment, drugs & fluids etc were & I had to rely solely on the staff around me to ‘go fetch’ as I kept barking orders.
      • This was a stark reminder of what Cliff Reid has been talking about for years, in the need to know and control your resuscitation environment.
      • I have subsequently spent my own time going through various parts of the department familiarising myself with the resus equipment and its location (not only in the operating theaters and recovery, but also on the ward resus-trollies where we attend medical emergencies).

3) Our ED training is great to fall back on…

      • Faced with a persistently hypotensive patient, I did what felt comfortable and what came naturally… I took ultrasound to the bedside.
      • Whilst this didn’t add anything new to the case, it backed up my suspicions at a time when I was doubting myself…

4) The patient in haemorrhagic shock can fall in a heap on induction…

      • Midaz/Fentanyl was all that was needed to reach reasonable sedation for induction.
      • I am left to ponder what would have happened if he was given a more ‘generous’ induction agent (eg. ketamine).

The Conclusion

Well, this is fortunately the boring part of the story. Our fella is delivered to ICU with stable haemodynamics, a temperature of 36.2*C & a normal pH/bicarb. His INR is 1.1 & Hb is 72.

He receives a further 2 units of RBCs overnight in the ICU & is extubated the following day.

By day 3 (post-op) he is back on the ward and makes a progressively uneventful recovery to hospital discharge.

 

So, there you go.

I’d love to hear peoples feedback and comments on this case.

 

About Christopher Partyka

Emergency Doc; interested in ultrasound, retrieval & medical education - #FOAMed

Comments

  1. Great case

    In retrospect, would you have called urology boss sooner?

    Reckon ketamine would’ve been fine….

  2. Alan Yan says:

    Thanks for sharing the case Nick. So often we see this in OT, unfortunately poor acknowledgement by surgical team often resulted in delay for re-OT.

    Synthetic starch (Voluven) still being the most commonly used colloids in most anaesthetic departments despite its negligible effect and possibly harm. Wished they were there to listen to Myburg’s talk!

    Ketamine would have been a better choice for induction in my opinion, however, its not always the induction of choice with almost all anesthetists.

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